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Browsing Center for Life Sciences by Subject "N-Methyl-D-aspartate"

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Status epilepticus enhances depotentiation after fully established LTP in an NMDAR-Dependent but GluN2B-independent manner

Guli, Xiati; Tokay, Tursonjan; Kirschstein, Timo; Köhling, Rüdiger (Neural Plasticity, 2015-11-15)

N-Methyl-D-aspartate (NMDA) receptor-dependent long-term potentiation (LTP) can be reversed by low-frequency stimulation (LFS) referred to as depotentiation (DP). We previously found GluN2B upregulated in CA1 neurons from ...