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Elevated levels of plasma collagen priple helix repeat-containing (CTHRC1) protein correlates with increased granul ocytopoiesis and higher rheumatoid arthritis disease severity score

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dc.contributor.author Myngbay, A.
dc.contributor.author Bexeitov, Y.
dc.contributor.author Adilbayeva, A.
dc.contributor.author Yevstratenko, B. P.
dc.contributor.author Aitzhanova, R. M.
dc.contributor.author Otarbayev, N. K.
dc.contributor.author Adarichev, V.A.
dc.date.accessioned 2016-05-30T10:35:00Z
dc.date.available 2016-05-30T10:35:00Z
dc.date.issued 2016-05
dc.identifier.citation A. Myngbay, Y. Bexeitov, A. Adilbayeva, B. P. Yevstratenko, R. M. Aitzhanova, N. K. Otarbayev, and V.A. Adarichev. 2016. Elevated levels of plasma collagen priple helix repeat-containing (CTHRC1) protein correlates with increased granul ocytopoiesis and higher rheumatoid arthritis disease severity score. Abstract book. 4 th International Scientific Conference “Regenerative medicine & healthy aging”. National Laboratory Astana, Nazarbayev University. http://nur.nu.edu.kz/handle/123456789/1521 ru_RU
dc.identifier.uri http://nur.nu.edu.kz/handle/123456789/1521
dc.description.abstract Monitoring and suppressing activity of the pathological granulating pannus tissue is essential for Rheumatoid Arthritis (RA) management. Synovial joints pain and tissue destruction is a derivative of the pannus activity. In a mouse model for RAwe found CTHRC1, a secreted and circulating protein, to be inducible in arthritis and correlated with the disease severity. We performed a clinical investigation to ascertain the role of CTHRC1 in the progression of the disease. ru_RU
dc.language.iso en ru_RU
dc.rights Attribution-NonCommercial-ShareAlike 3.0 United States *
dc.rights.uri http://creativecommons.org/licenses/by-nc-sa/3.0/us/ *
dc.subject Rheumatoid Arthritis ru_RU
dc.title Elevated levels of plasma collagen priple helix repeat-containing (CTHRC1) protein correlates with increased granul ocytopoiesis and higher rheumatoid arthritis disease severity score ru_RU
dc.type Abstract ru_RU


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